It’s not just gang culture the US army is exporting.
Wired News is reporting on the superbug that has evolved (but which was first blamed on the ‘dirty’ Iraqi soil) in Iraq military field hospitals and which has since been exported to hospitals all over Europe:
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Since OPERATION Iraqi Freedom began in 2003, more than 700 US soldiers have been infected or colonized with Acinetobacter baumannii. A significant number of additional cases have been found in the Canadian and British armed forces, and among wounded Iraqi civilians. The Armed Forces Institute of Pathology has recorded seven deaths caused by the bacteria in US hospitals along the evacuation chain. Four were unlucky civilians who picked up the bug at Walter Reed Army Medical Center in Washington, DC, while undergoing treatment for other life-threatening conditions. Another was a 63-year-old woman, also chronically ill, who shared a ward at Landstuhl with infected coalition troops.
Behind the scenes, the spread of a pathogen that targets wounded GIs has triggered broad reforms in both combat medical care and the Pentagon’s networks for tracking bacterial threats within the ranks. Interviews with current and former military physicians, recent articles in medical journals, and internal reports reveal that the Department of Defense has been waging a secret war within the larger mission in Iraq and Afghanistan – a war against antibiotic-resistant pathogens.
Acinetobacter is only one of many bacterial nemeses prowling around in ICUs and neonatal units in hospitals all over the world. A particularly fierce organism known as MRSA – methicillin-resistant Staphylococcus aureus – infects healthy people, spreads easily, and accounts for many of the 90,000 fatal infections picked up in US hospitals each year. Another drug-resistant germ on the rise in health care facilities, Clostridium difficile, moves in for the kill when long courses of antibiotics have wiped out normal intestinal flora.
Forerunners of the bug causing the military infections have been making deadly incursions into civilian hospitals for more than a decade. In the early 1990s, 1,400 people were infected or colonized at a single facility in Spain. A few years later, particularly virulent strains of the bacteria spread through three Israeli hospitals, killing half of the infected patients. Death by acinetobacter can take many forms: catastrophic fevers, pneumonia, meningitis, infections of the spine, and sepsis of the blood. Patients who survive face longer hospital stays, more surgery, and severe complications.
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Until a few years ago, most strains could be dispatched with a wide variety of drugs. For the most tenacious infections, doctors could rely on a family of ultrabroad spectrum antibiotics called carbapenems. But strains of acinetobacter are emerging now that are immune to every known remedy. Multidrug – resistant pathogens are an epidemiologist’s nightmare – reminders of the dark ages when millions of people died every year of runaway infections.
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And they’re spreading fast. A major outbreak in Chicago two years ago infected 81 patients, killing at least 14. Arizona health officials tracked more than 200 infections in state hospitals early last year. Doctors at Vanderbilt University Medical Center in Tennessee used to see an infection or two every year; now it’s one or more a month. “These bacteria are developing very, very quickly,” says CDC epidemiologist Arjun Srinivasan, who has been consulting with the DOD about the military outbreak. “The bad news is that we’re many years away from having new drugs to treat them. It should be a call to arms.”
